Yup, in other words, treating a trauma with a benzo makes the long term outcome worse. It seems to disrupt the bodies natural healing process and increases startle and trauma responses over time.
Again, trusting the body to heal itself seems to be something no one wants to do.
This is an abstract from PubMed. It’s a study done on rats. I realize humans aren’t rats but I know I’ve observed similar outcomes in human beings and I think most of you would agree the drugs never helped us and often made us worse. Benzos are also horribly addictive.
Unfortunately it’s being proven that all these drugs raise relapse rates. I have studies that indicate such if you’d like I’ll share. Here is Vera Sherav’s take on another study with antipsychotics showing long-term use increases relapse rates. I’ve got the PDF file of the original study if you want it.
And below is the abstract of the study done with Xanax that I will dare to say can be generalized with all benzos and I’d go as far as to say the rest of the psychotropic drugs.
BACKGROUND: In light of clinical reports suggesting that early benzodiazepine administration interferes with long-term recovery from traumatic stress, a prospective animal model for PTSD was employed to assess the short- and long-term effects of a brief course of alprazolam following stress exposure.
Method: Animals exposed to stress were treated either 1 h or 7 days later with alprazolam or vehicle for 3-days. Outcome measures included behavior in the elevated plus-maze (EPM) and acoustic startle response (ASR) tests 30 days after initial exposure and freezing behavior upon exposure to a trauma-cue on day 31. One group was repeatedly exposed to the triggering trauma shortly before and after treatment and assessed as above. Circulating corticosterone levels were assessed 4 h after initiation of alprazolam and post-treatment. Pre-set cut-off behavioral criteria classified exposed animals according to their EPM and ASR response-patterns into ‘extreme’, ‘minimal,’ or ‘partial’ behavioral response for analysis of prevalence rates. Results: Immediate alprazolam treatment was effective in alleviating anxiety at day 4. No observable anxiolytic effects remained at day 30. Immediate alprazolam also resulted in significantly greater freezing response to trauma-cue exposure and in extreme responses to double-exposure. Corticosterone levels were significantly suppressed by alprazolam during treatment and rebounded after cessation.
Conclusion: A brief course of alprazolam in the immediate aftermath of stress-exposure is associated with less favorable responses to additional stress-exposure later on. Alprazolam was associated with a significant attenuation of the HPA-response, suggesting a possible link between initial HPA-axis response disruption and the subsequent unfavorable outcomes.